All publications may be found here.

Low-Intensity Ultrasound-Induced Anti-inflammatory Effects Are Mediated by Several New Mechanisms Including Gene Induction, Immunosuppressor Cell Promotion, and Enhancement of Exosome Biogenesis and Docking.

Interleukin-10 Inhibits Bone Marrow Fibroblast Progenitor Cell-Mediated Cardiac Fibrosis in Pressure-Overloaded Myocardium.

Therapeutic inhibition of miR-375 attenuates post-myocardial infarction inflammatory response and left ventricular dysfunction via PDK-1-AKT signalling axis.

Restoration of Hydrogen Sulfide Production in Diabetic Mice Improves Reparative Function of Bone Marrow Cells.

c-kit+ Cardiac Stem Cells: Spontaneous Creation or a Perplexing Reality.

N-Acetyl cysteine protects diabetic mouse derived mesenchymal stem cells from hydrogen-peroxide-induced injury: A novel hypothesis for autologous stem cell transplantation.

IL-10 Accelerates Re-Endothelialization and Inhibits Post-Injury Intimal Hyperplasia following Carotid Artery Denudation.

More Than Tiny Sacks: Stem Cell Exosomes as Cell-Free Modality for Cardiac Repair.

Interleukin-10 inhibits chronic angiotensin II-induced pathological autophagy.

Negative Regulation of miR-375 by Interleukin-10 Enhances Bone Marrow-Derived Progenitor Cell-Mediated Myocardial Repair and Function After Myocardial Infarction.

Enhanced Cardiac Regenerative Ability of Stem Cells After Ischemia-Reperfusion Injury: Role of Human CD34+ Cells Deficient in MicroRNA-377.

Sirtuin-6 deficiency exacerbates diabetes-induced impairment of wound healing.

Hrd1 and ER-Associated Protein Degradation, ERAD, are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes.

Embryonic stem cell-derived exosomes promote endogenous repair mechanisms and enhance cardiac function following myocardial infarction.

Stem Cells and Cardiac Repair.

Mesenchymal Stem Cells Pretreated with HGF and FGF4 Can Reduce Liver Fibrosis in Mice.

Cardiac progenitor cells engineered with βARKct have enhanced β-adrenergic tolerance.

Rejuvenation of human cardiac progenitor cells with Pim-1 kinase.

Preconditioning diabetic mesenchymal stem cells with myogenic medium increases their ability to repair diabetic heart.

Mesenchymal stem cells and Interleukin-6 attenuate liver fibrosis in mice.

β-Adrenergic regulation of cardiac progenitor cell death versus survival and proliferation.

Human cardiac progenitor cells engineered with Pim-I kinase enhance myocardial repair.

Mesenchymal stem cells conditioned with glucose depletion augments their ability to repair-infarcted myocardium.

Bone marrow derived mesenchymal stem cells from aged mice have reduced wound healing, angiogenesis, proliferation and anti-apoptosis capabilities.

Nitric oxide augments mesenchymal stem cell ability to repair liver fibrosis.

Pim-1 kinase inhibits pathological injury by promoting cardioprotective signaling.

Mitochondrial translocation of Nur77 mediates cardiomyocyte apoptosis.

Myocardial AKT: the omnipresent nexus.

Repair of senescent myocardium by mesenchymal stem cells is dependent on the age of donor mice.

Nucleolar stress is an early response to myocardial damage involving nucleolar proteins nucleostemin and nucleophosmin.

Enhanced hepatic differentiation of mesenchymal stem cells after pretreatment with injured liver tissue.

Growth factor preconditioning increases the function of diabetes-impaired mesenchymal stem cells.

Autosomal recessive congenital cataract in consanguineous Pakistani families is associated with mutations in GALK1.

Lin-c-kit(+) BM-derived stem cells repair Infarcted Heart.

A new locus for autosomal recessive congenital cataract identified in a Pakistani family.

IGF-1 and G-CSF complement each other in BMSC migration towards infarcted myocardium in a novel in vitro model.

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Temple University

Lewis Katz School of Medicine